UVEYT (uveitis; late lat. uvea a choroid of an eye + - itis) — an inflammation of a choroid (vascular, or uveal, a path) eyes.
Due to the features of an anatomic structure of a choroid of an eye, in particular separate blood supply and an innervation of its front department — irises (see), a ciliary body (see) and back — an idiovascular cover, or a horioidea (see the Choroid of an eye), inflammatory processes, as a rule, develop in them separately; an inflammation front otdelya a choroid of N and z t in and go t both r and d about a cycle and t about m (see), or to lobbies At., back department of a choroid — a choroiditis (see), or a back uveitis. In rare instances the inflammation extends to all vascular highway of an eye — iridotsiklokhorioidit (pan-uveitis). Since the end of the 50th years allocate as an independent form an inflammation of a flat part of a ciliary body (a ciliary circle) and the extreme periphery of an idiovascular cover — peripheral At. On Woods's classification (A. Woods, 1956) distinguish granulematozny At., at to-rykh the inflammatory center has character of a granuloma (see) with a perifocal inflammation, and negranulematozny, at to-rykh an inflammation has diffuse character.
Distinguish the exogenous uveites developing at the getting wounds of an eye, at a perforated helcoma and other damages of an eye and the endogenous uveites arising at various general diseases.
The leading role in a pathogeny At. play immune mechanisms. Depending on immunol. reactions of a choroid are distinguished atopic At. (see the Atopy), connected with effect of exogenous allergens (pollen of plants, foodstuff, etc.); anaphylactic At. (see the Anaphylaxis), caused by development of allergic reaction to introduction to an organism of immune serum; autoallergichesky At. (see the Autoallergy), at to-rykh allergen the pigment of a choroid or squirrels of a crystalline lens is (fakogenny At.); microbic and allergic (see. An infectious allergy), the infections developing in the presence in an organism of the centers.
Identification etiol. a factor at At. presents great difficulties. In large part cases an origin At. remains obscure.
Features of pathological anatomy, a clinical picture, diagnosis, treatment and the forecast of an inflammation of an iris and a ciliary body are stated in article an iridocyclitis (see), an idiovascular cover — in article a choroiditis (see); the forms of a uveitis which are not described by independent articles are included below.
Both r and d about c and to l about x about r and about and d and t (a pan-uveitis, generalized at in e and r) — the most severe form At., it is observed in two versions: acute and chronic.
Acute iridotsiklokhorioidit arises as a result of a drift of pathogenic microorganisms in capillaries of a choroid or a retina. It is characterized by sharp eye pains, decrease in sight.
Inflammatory process (as a rule, purulent) quickly extends to an iris and a ciliary body with frequent development of abscess of a vitreous (see Endoftaljmit). At especially heavy current the purulent inflammation of all covers of an eyeglobe is observed (see Panoftal-Meath). Treatment of an acute iridotsiklo-choroiditis consists in introduction of antibiotics intramusculary, under a conjunctiva, retrobulbarno, in an anterior chamber of an eye, a vitreous; make a vitreoek-tomiya (see the Vitreous), at impossibility to keep an eye — evisceration of an eye (see). During the progressing of process the forecast adverse.
X roniches the cue ir and to CEC of l about x about r io can-dit also have a herpetic, brucellous, tubercular etiology, meets at a sarcoidosis (see), Vogt's syndrome — Koyanagi (see Vogt — Koyanagi a syndrome). Develops within several years with frequent aggravations. As a rule, affects both eyes. Formation of considerable opacities of the vitreous body, exudative centers in a choroid is characteristic that leads to decrease in sight. Treatment is complex, includes the hyposensibilizing influence, use of antibiotics, antiviral drugs, immunodepressants, in late stages — excision shvart a vitreous.
During the progressing of process — the forecast serious, in many cases develops an atrophy of an eyeglobe.
Periferich with to and y at in e-and the t develops hl. obr. aged from 20 up to 35 years; affects more often both eyes.
At the beginning of a disease moderate decrease in sight, unsharply expressed photophobia is noted. At biomicroscopy of an eye (see) small powdery opacities of the vitreous body come to light. Gradually disorganization of a vitreous and local accumulation of exudate in the field of the gear line develops; exudate in a vitreous of white or grayish-white color, dense, is well delimited, gradually spreads to the area of a ciliary body.
Sometimes find small corneal precipitated calcium superphosphates and front synechias. In late stages amotio of a vitreous, education rough shvart is possible that it is followed by considerable decrease in sight. As complications peripheral At. can develop a cataract (see), secondary glaucoma (see), secondary dystrophy of a retina in macular area (see Retinopathies), a papilledema. Amotio of a retina, a retinal apoplexy belong to rare complications retnnoshizis (see the Retina). Treatment peripheral At. carry out in a complex. by the same principles, as treatment of an iridocyclitis (impact on a source of an infection and a sensitization. on immune mechanisms p on vospalitelnyn process in a choroid). In detail — see//r//rtouii K.'i of Thursday.
the Forecast at timely and
correct treatment at the majority
Bol mi y \, | aroi (a riya tny.
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