SIDEROSIS (siderosis: Greek sideros iron + — osis) — the pneumoconiosis developing under the influence of the dust containing iron and its connections. Occurs at miners at extraction of the red iron ore containing up to 70% of iron (hematite, natural iron oxide), and at working foundry workshops, polishers, engravers, electric welders.
S.'s existence as independent disease of many researchers is denied. Often it is about a siderosilicosis or a sili-kosideroza, the current to-rykh depends on whether prevail in dust of compound of iron or free quartz. At the considerable content of quartz the disease comes nearer to to a silicosis (see). Impurity of dust of iron can reduce fibrogenny effect of free silicon dioxide that causes more favorable disease in comparison with a silicosis.
The maximum allowable concentration for iron oxide with impurity of oxides of manganese to 3% makes 6 mg/m3, for iron oxide with impurity of fluoric or 3 — 6% of manganese connections — 4 mg/m3, for iron and nickel is agglomerate — 4 mg/m3.
Morphologically distinguish red and black S. Krasny S. there is owing to influence of dust, oxidiferous gland. S., macroscopically easy at this look, are increased in volume, a dense consistence and painted in burovatokrasny color. The black siderosis develops at influence of the dust containing ferrous oxide or carbonic and its phosphate connections. Lungs at black S. of black color and by outward do not differ from those at an anthracosis (see. Pneumoconiosis ).
At gistol. a research in lungs at S. it is found moderately expressed sclerosis (see), the submiliary and miliary small knots of rounded, oval, irregular shape consisting of accumulations of closely lying dust cells filled with the particles of dark-brown color (dust of iron) giving positive reaction to iron (see. Perlsa way ). Between dust cells not numerous collagenic fibers, flour particles of iron and single capillaries are found. Accumulations of dust cells often come to light also around small vessels, and the content of collagenic fibers in these sites is small. In limf, nodes the diffusion sclerosis comes to light, particles of dust are found.
A wedge, a picture at S. extremely scanty. The page can be shown by poorly expressed symptoms bronchitis (see) and to be followed by insignificant functional disturbances. On roentgenograms of lungs the abundance of the smallest dot intensive shadows, as a rule, not merging in conglomerates is often observed, the pulmonary drawing is usually a little strengthened. Discrepancy between scanty a wedge, symptoms and clear rentgenol. is explained changes by impermeability of ferriferous dust for x-ray emission. Disease is usually high-quality; progressing of process at the termination of contact with dust usually is not observed, in nek-ry cases even regression of process is possible. Disease and the forecast are made heavier in cases of accession of tuberculosis and banal infection.
Treatment, examination of working capacity and prevention the same, as at others pneumoconiosis (see).
Bibliography: P. P's cursors. Pneumoconiosis, M., 1965; To about in N and c to and y M. A. Klinika of a pneumoconiosis, JI., 1963, bibliogr.; Occupational diseases, under the editorship of A. A. Letavet, etc., page 435, M., 1973; With t r at to about in A. I. and Serov V. V. Pathological anatomy, page 479, M., 1979; P a r k e s W. R. Occupational lung disorders, L., 1974.
H. A. Senkevich; G. M. Mogilevsky (stalemate. An.).